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The expression of the Ctnnb1 gene showed no significant difference between 0 mol/L and 20 mol/L anwuligan treatment groups (not shown)

Treatment benefit is maximal at 16 mg/day as monotherapy [22]

The transition from observing natural NPY as a biomarker to using exogenous NPY as a treatment involves assumptions about dose-response relationships that have not been fully validated in human trials

A future direction would be to include a healthy control population to ensure differentiation between OI and healthy control groups in comparison to concussion

Imagine glutamine is an amino acid, which means its a building block for protein in the body